Does anyone know what my ultrasound shows?

keangkong

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Does anyone know what my ultrasound from today shows? I don't see the endocrinologist until Friday of next week. This is what the radiologist wrote:

US Abdomen Complete

REASON FOR EXAM
: Metabolic dysfunction-associated steatotic liver disease (MASLD)

COMPARISON: None

TECHNIQUE: Transabdominal imaging with grayscale, color Doppler and duplex evaluation.

FINDINGS:

Liver: Liver is normal in size. Liver parenchyma is diffusely increased in echogenicity. Smooth surface contour. No solid liver mass. Main portal vein is patent with appropriate direction flow.

Gallbladder: Gallbladder is normal in size without stones or sludge. No wall thickening. No pericholecystic fluid. Sonographic Murphy's sign is negative. No intrahepatic bile duct dilation.

Pancreas: Visualized pancreatic head and neck are normal in echogenicity.

Kidneys: Kidneys are normal in size and echogenicity. No hydronephrosis. No solid mass.

Aorta/ IVC: Normal in size without aneurysm.

Spleen: Normal in size and echogenicity.

Other: No ascites.

Measurements:
Liver Size: 16.8 cm
Spleen Size (greatest dimension): 11.0 cm
CBD: 2 mm
Right Kidney: 11.7 x 6.5 x 6.2 cm
Left Kidney: 11.9 x 7.7 x 5.7 cm

IMPRESSION:
1. Hepatic steatosis.
2. Otherwise no acute process identified in the imaged abdomen.
 
Does anyone know what my ultrasound from today shows? I don't see the endocrinologist until Friday of next week. This is what the radiologist wrote:

US Abdomen Complete

REASON FOR EXAM
: Metabolic dysfunction-associated steatotic liver disease (MASLD)

COMPARISON: None

TECHNIQUE: Transabdominal imaging with grayscale, color Doppler and duplex evaluation.

FINDINGS:

Liver: Liver is normal in size. Liver parenchyma is diffusely increased in echogenicity. Smooth surface contour. No solid liver mass. Main portal vein is patent with appropriate direction flow.

Gallbladder: Gallbladder is normal in size without stones or sludge. No wall thickening. No pericholecystic fluid. Sonographic Murphy's sign is negative. No intrahepatic bile duct dilation.

Pancreas: Visualized pancreatic head and neck are normal in echogenicity.

Kidneys: Kidneys are normal in size and echogenicity. No hydronephrosis. No solid mass.

Aorta/ IVC: Normal in size without aneurysm.

Spleen: Normal in size and echogenicity.

Other: No ascites.

Measurements:
Liver Size: 16.8 cm
Spleen Size (greatest dimension): 11.0 cm
CBD: 2 mm
Right Kidney: 11.7 x 6.5 x 6.2 cm
Left Kidney: 11.9 x 7.7 x 5.7 cm

IMPRESSION:
1. Hepatic steatosis.
2. Otherwise no acute process identified in the imaged abdomen.
Chatgpt gave me this :

Hi keangkong,
Thanks for sharing your results. From what you posted, it looks like the main finding is hepatic steatosis (fatty liver), which is pretty common, especially with weight fluctuations or metabolic changes — including when using GLP-1 medications. The good news is that everything else (gallbladder, kidneys, spleen, pancreas, vessels) appears normal, and there are no signs of acute issues like masses, stones, or obstructions.

Of course, it's always best to go over the full report with your doctor, but based on the info you shared, it doesn't seem alarming. Managing fatty liver usually involves weight management, healthy diet, and sometimes adjusting medications. You're definitely not alone with this finding!

Hope everything goes well with your follow-up.
 
Congrats. It means you are a healthy fat ass like me. Keep doing what you are doing to get healthy. Years of pizza chicken wings and beer finally took its toll. At least thats what did it for me. It can take a long time to dry out the fatty liver. I practice Chinese medicine in real life, so I'm not a "real" MD.
 
In other way of looking at it. Its the earliest but reversible path to cirrhosis of the liver. Alt and ast were probably elevated which prompted the Doc to send for an ultrasound. If the numbers were more than double the normal range, it warrants a look on ultrasound for confirmation, otherwise they just wanted money.
 
r/AskDocs on Reddit often fields questions like this.

However, the docs there will also tell you to not try and interpret the results yourself, and just wait to have a talk with your provider. You can make yourself crazy trying to figure out what it really means. All kinds of imaging reports stuff that's not really meaningful and it's easy to worry needlessly.

AskDocs is a really interesting place to read, I look at it often.
 
Glutathione (highly endorsed by @AndyPanda) may also help with liver function:

Google Gemini said:
Yes, glutathione can potentially help with liver function in several ways:

Antioxidant and Detoxification:
  • Glutathione is a powerful antioxidant that helps neutralize harmful free radicals in the liver, reducing oxidative stress and protecting liver cells from damage.

  • It plays a crucial role in the liver's detoxification processes, helping to bind with toxins and facilitate their removal from the body. This includes processing metabolic waste, pollutants, and certain drugs.
Specific Liver Conditions:
  • Fatty Liver Disease: Studies have shown that glutathione may improve liver enzyme levels (like ALT), reduce fat accumulation, and lower triglycerides and ferritin levels in individuals with both alcoholic and non-alcoholic fatty liver disease (NAFLD).

  • Liver Injury: Glutathione supplementation has shown potential in attenuating liver injury caused by various factors.

  • Overall Liver Health: By supporting detoxification and reducing oxidative stress, glutathione helps maintain the liver's overall function and health.
How it Works:
  • Glutathione is involved in essential processes within the liver, including the breakdown of free radicals and the processing of toxins so they can be eliminated.

  • It helps maintain the redox balance in the liver, which is crucial for proper cellular function.
Important Considerations:
  • While the body produces glutathione, levels can be reduced due to poor diet, stress, environmental toxins, and aging.

  • Glutathione can be supplemented orally, intravenously, or through inhalation.8 Oral bioavailability can be a concern, with liposomal forms potentially offering better absorption. N-acetylcysteine (NAC) is a precursor to glutathione and can also help boost its levels.

  • It's always best to consult with a healthcare professional before starting any glutathione supplementation to determine the appropriate form, dosage, and to ensure it's suitable for your individual health needs.
 
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This is all bordering (if not crossing the line into) medical advice and I would highly advise against it.
I suggest not seeking medical interpretations from a forum like this.
 
This is all bordering (if not crossing the line into) medical advice and I would highly advise against it.
I suggest not seeking medical interpretations from a forum like this.
Counterpoint: from what I can tell none of us have ever been wrong in anything we have said on this forum, even when we are in disagreement on facts. Where better to get sound advice than a place where every remark contains ultimate, unbending truth?
 
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Reta actually reduces fatty liver content. The chart is from this article: https://www.nature.com/articles/s41591-024-03018-2 I'm not a doctor and this is not medical advice. When working with unapproved peps we are all self-experimenting. I will say that personally, my bloodwork from August showed a borderline fatty liver. My bloodwork from January showed the numbers dropped to half and were well within the normal range. Something to research. Probably not something to discuss with your doctor since Reta is not approved.
 
View attachment 6605
Reta actually reduces fatty liver content. The chart is from this article: https://www.nature.com/articles/s41591-024-03018-2 I'm not a doctor and this is not medical advice. When working with unapproved peps we are all self-experimenting. I will say that personally, my bloodwork from August showed a borderline fatty liver. My bloodwork from January showed the numbers dropped to half and were well within the normal range. Something to research. Probably not something to discuss with your doctor since Reta is not approved.
I used to work for a Virginia state Behavioral Health agency as a department head, under a guy who insisted people call him Doctor. He was the COO of the campus and was only involved administrative processes. Had a phony MS from a diploma mill named Kennedy Western University, and a phony PhD from a diploma mill named Warren National University. He was making about $300k per year. Still does. Someone reported him to the State OIG and they did absolutely nothing about it. Apparently, calling yourself Doctor is protected speech. You just can’t claim to be a licensed practitioner. He has since been promoted.

I think you should DEFINITELY call yourself a Doctor. Just leave in the disclaimer that it’s not medical advice.
 
This is all bordering (if not crossing the line into) medical advice and I would highly advise against it.
I suggest not seeking medical interpretations from a forum like this.
Speculating about the results of something he has already said he is talking to his doctor about seems to be significantly less risky than the overwhelming majority of what happens on this forum. We spend a good amount of time talking about research chemicals that have limited or no human trials done.

Like, we've got people going around advocating for slu-pp-332 with no trials or stuff that is in the early stages of phase-1/phase-2 trials. Even promoting reta, which personally I infer as just fine based on phase-3 trials being extended, etc., would probably be considered a way riskier proposition by an actual medical professional than someone going "anyone know what this means?" and us going "i dunno man looks like maybe you have fatty liver based on it saying you have fatty liver"
 
Liver news:



(Maybe mostly from the weight/fat loss, but still impressive.)
Maybe not mostly from weight loss though-- gleaning through that study (which unfortunately was only for oral glutathione, but still...) it mentioned, and I'm severely paraphrasing, that fatty liver condition left liver susceptible to injury from among other thing, cytokines. Lowering overall inflammation, as glp-1's do almost immediately, lowers cytokine levels, lessening the progression to fibrosis, cirrhosis.
 

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