Dumb question about Tessa and Bloods.

Gr33dyOctopus

Gr33dyOctopus

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Alright, I've been in a few threads for GH and definitely not the sharpest knife in the box. I have HGH, I have Tesa, and would like to start one or the other up but very concerned about blood sugar.

When i started this glp1 experiment I had a 5.7 ac1. It is now 5.3. Really dont want to be in diabetes territory but id also like the 1 to 2iu benefits gh might provide me, at least I wanna try it.

My question is bloodwork. Can I just get an Amazon finger stick machine and go from there? I asked for further bloodwork from my doctor and they told me " We just checked a couple months ago, not happening "

And yes, I've been around here long enough I should know the answer to this question, but I dont. Whats the best and easiest way to make sure using gh or tessa isnt going to kill me?
 
CNCCurrency said:
Why?
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Tesa is a GH secretagogue that supposedly targets visceral fat loss. The GH pulsing resulting from Tesa should be bearable, but once bulking phase begins, and calorific deficit is no longer as atrocious, pure GH can be used to make use of its anabolic actions on all pathways. After all GH acts on hunger receptors too.
 
Meritocrat said:
Tesa is a GH secretagogue that supposedly targets visceral fat loss. The GH pulsing resulting from Tesa should be bearable, but once bulking phase begins, and calorific deficit is no longer as atrocious, pure GH can be used to make use of its anabolic actions on all pathways. After all GH acts on hunger receptors too.
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Tesa is a GH secretagogue that supposedly targets visceral fat loss. That doesn't make sense to me, and hgh wouldn't be as effective?
 
CNCCurrency said:
Tesa is a GH secretagogue that supposedly targets visceral fat loss. That doesn't make sense to me, and hgh wouldn't be as effective?
Click to expand...
That's why I never got that claim myself, but what I can comprehend is that the secretagogues make a more typical GH release in the form of a pulse of somatotropin release, while direct GH supplementation can end up with a more artificial, longer-lasting presence, thereby amplifying the effects of GH, but also elevating the likelihood of sides. At least, that's how I understand it. I have heard more about sides from actual GH usage while I have heard of water retention as the most common side with secretagogues.
 
Nearly everyone on this forum is on a GLP drug. While not tested in a human clinical trial it is a fairly safe bet that the glucose lowering effect of glp drugs will have a stronger effect in reducing blood glucose than low doses of tesa or gh will have in increasing it. I assume you are on reta or tirz?

There is a reason to be a bit more careful in your case, though the effect of glp drugs will still be the same, the initial hb1ac was 5.7 which is at the bottom of the impaired glucose tolerance or pre diabetes range, the more recent one is fine at 5.1. Just means checking sugars a bit more often and hb1ac occasionally , and get igf-1 checked. I would suggest taking tesa or hgh without a glp drug , with possible pre diabetes is not a great idea.
 
lessthanhalf said:
Nearly everyone on this forum is on a GLP drug. While not tested in a human clinical trial it is a fairly safe bet that the glucose lowering effect of glp drugs will have a stronger effect in reducing blood glucose than low doses of tesa or gh will have in increasing it. I assume you are on reta or tirz?

There is a reason to be a bit more careful in your case, though the effect of glp drugs will still be the same, the initial hb1ac was 5.7 which is at the bottom of the impaired glucose tolerance or pre diabetes range, the more recent one is fine at 5.1. Just means checkingdrug sugars a bit more often and hb1ac occasionally , and get igf-1 checked. I would suggest taking tesa or hgh without a glp , with possible pre diabetes is not a great idea.
Click to expand...
I would suggest taking tesa or hgh without a glp
Confused Thinking GIF by MOODMAN
 
Gr33dyOctopus said:
Alright, I've been in a few threads for GH and definitely not the sharpest knife in the box. I have HGH, I have Tesa, and would like to start one or the other up but very concerned about blood sugar.

When i started this glp1 experiment I had a 5.7 ac1. It is now 5.3. Really dont want to be in diabetes territory but id also like the 1 to 2iu benefits gh might provide me, at least I wanna try it.

My question is bloodwork. Can I just get an Amazon finger stick machine and go from there? I asked for further bloodwork from my doctor and they told me " We just checked a couple months ago, not happening "

And yes, I've been around here long enough I should know the answer to this question, but I dont. Whats the best and easiest way to make sure using gh or tessa isnt going to kill me?
Click to expand...
I'd monitor more than just blood sugar. A finger stick meter is a good, inexpensive starting point, but I'd also get a baseline IGF1 and recheck it after 4–8 weeks. Whether you're using GH or tesa, IGF1 gives you an idea of how much GH effect you're actually getting, while fasting glucose (or Ideally and not too expensive a CG) , tells you whether it's affecting your blood sugar.
With an A1c that has gone from 5.7% to 5.3%, you're starting from a pretty good place 😉 . I'd just watch the trends rather than obsessing over any single reading. Neither GH nor tesamorelin is automatically blood sugar neutral, but monitoring IGF1 and glucose together should give you a pretty good picture of how you're responding.
 
Use Goodlabs for bloodwork and run your results through AI. Doctors are a thing of the past.
 
Jfrick11 said:
I'd monitor more than just blood sugar. A finger stick meter is a good, inexpensive starting point, but I'd also get a baseline IGF1 and recheck it after 4–8 weeks. Whether you're using GH or tesa, IGF1 gives you an idea of how much GH effect you're actually getting, while fasting glucose (or Ideally and not too expensive a CG) , tells you whether it's affecting your blood sugar.
With an A1c that has gone from 5.7% to 5.3%, you're starting from a pretty good place 😉 . I'd just watch the trends rather than obsessing over any single reading. Neither GH nor tesamorelin is automatically blood sugar neutral, but monitoring IGF1 and glucose together should give you a pretty good picture of how you're responding.
Click to expand...
Mine is 5.1 using hgh and reta. So not an issue! Just because others are uncontrolled they hate on hgh every chance the get on here.
 
Gr33dyOctopus said:
???
Click to expand...
All anabolic effects of GH are primarily due to IGF-1, and one's muscle growth is limited by somite-originated satellite cell activation. Here's the AI explanation.

The primary hormones impacting your satellite cell pool include:

Testosterone and Androgens
  • Direct Proliferation: Testosterone directly binds to androgen receptors on satellite cells. This action forces them to multiply rapidly and increases the total absolute number of satellite cells available for muscle repair.
Insulin-like Growth Factor-1 (IGF-1) & MGF
  • The Activation Switch: Localized IGF-1 is found directly inside the satellite cell microenvironment. It acts as the primary "on" switch following mechanical damage. [1, 2]
  • Differentiation: It forces activated satellite cells to transform into myoblasts, which are the precursor cells that physically fuse together to fix tears. [1, 2]
  • Mechano-Growth Factor: Mechanical stress converts IGF-1 into MGF, extending the cell's protein synthesis window for up to 72 hours post-exercise. [1]

Growth Hormone (GH)
  • Mobilization: Growth Hormone does not usually act on the cells alone. Instead, it works by stimulating systemic IGF-1 production.
Hence, I would prefer cycling IGF and MGF when looking for anabolic effects... with TRT of course... No insulin resistance, joint issues, bloat, etc.
 
Meritocrat said:
All anabolic effects of GH are primarily due to IGF-1, and one's muscle growth is limited by somite-originated satellite cell activation. Here's the AI explanation.

The primary hormones impacting your satellite cell pool include:

Testosterone and Androgens
  • Direct Proliferation: Testosterone directly binds to androgen receptors on satellite cells. This action forces them to multiply rapidly and increases the total absolute number of satellite cells available for muscle repair.
Insulin-like Growth Factor-1 (IGF-1) & MGF
  • The Activation Switch: Localized IGF-1 is found directly inside the satellite cell microenvironment. It acts as the primary "on" switch following mechanical damage. [1, 2]
  • Differentiation: It forces activated satellite cells to transform into myoblasts, which are the precursor cells that physically fuse together to fix tears. [1, 2]
  • Mechano-Growth Factor: Mechanical stress converts IGF-1 into MGF, extending the cell's protein synthesis window for up to 72 hours post-exercise. [1]

Growth Hormone (GH)
  • Mobilization: Growth Hormone does not usually act on the cells alone. Instead, it works by stimulating systemic IGF-1 production.
Hence, I would prefer cycling IGF and MGF when looking for anabolic effects... with TRT of course... No insulin resistance, joint issues, bloat, etc.
Click to expand...
Meritocrat said:
All anabolic effects of GH are primarily due to IGF-1, and one's muscle growth is limited by somite-originated satellite cell activation. Here's the AI explanation.

The primary hormones impacting your satellite cell pool include:

Testosterone and Androgens
  • Direct Proliferation: Testosterone directly binds to androgen receptors on satellite cells. This action forces them to multiply rapidly and increases the total absolute number of satellite cells available for muscle repair.
Insulin-like Growth Factor-1 (IGF-1) & MGF
  • The Activation Switch: Localized IGF-1 is found directly inside the satellite cell microenvironment. It acts as the primary "on" switch following mechanical damage. [1, 2]
  • Differentiation: It forces activated satellite cells to transform into myoblasts, which are the precursor cells that physically fuse together to fix tears. [1, 2]
  • Mechano-Growth Factor: Mechanical stress converts IGF-1 into MGF, extending the cell's protein synthesis window for up to 72 hours post-exercise. [1]

Growth Hormone (GH)
  • Mobilization: Growth Hormone does not usually act on the cells alone. Instead, it works by stimulating systemic IGF-1 production.
Hence, I would prefer cycling IGF and MGF when looking for anabolic effects... with TRT of course... No insulin resistance, joint issues, bloat, etc.
Click to expand...
Well if Ai said it, its gosple! Oh brother not another one of those sheep.
 
This thread is giving me cancer. Random leg, nonsense about tesa being superior than hgh at visceral fat. One talking about glps reduce glucose than recommends not using a glp with hgh.

Yes, finger prick is more than adequate to check for rise of fasting glucose when using either hgh or tesa.

If using reta, visceral fat is targeted hard, anyways. Btw on hgh my visceral fat which was low based on dexa, decreased another 1lb while putting on 20lbs in a bulk. Between 3-4 iu and fasting glucose was unchanged.
 
gusterbuster said:
This thread is giving me cancer. Random leg, nonsense about tesa being superior than hgh at visceral fat. One talking about glps reduce glucose than recommends not using a glp with hgh.

Yes, finger prick is more than adequate to check for rise of fasting glucose when using either hgh or tesa.

If using reta, visceral fat is targeted hard, anyways. Btw on hgh my visceral fat which was low based on dexa, decreased another 1lb while putting on 20lbs in a bulk. Between 3-4 iu and fasting glucose was unchanged.
Click to expand...
I take it you missed the point on the leg? I agree with you on the majority
 
CNCCurrency said:
I take it you missed the point on the leg? I agree with you on the majority
Click to expand...
I get the point, but as per the initial post, it is random as not pertaining to glucose levels.

And it in my head, adding random leg on top of this trainwreck was hysterical.

And i dont really think tesa or hgh protects muscle that well in a cut. It adds water to the muscle which will show on dexa as increased lean mass. Been on 4iu hgh on this cut and strength loss is as expected as if I wasnt using any. I mean hgh is studied at 18iu for muscle wasting in AIDS patients so doubt they would use that much if 4 iu was sufficient as prescription serostim is more than a mortgage at that dose.
 
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