I have had all that run but would have to check my numbers again.
Adding on Levothyroxine?
- Thread starter Samco12
- Start date
roxygirl
GLP-1 Novice
You shouldn’t you should always do your own research. Knowledge is power. Endo’s are lazy in many cases and narrow minded. They treat the masses with Levothyroxin or Synthroid with focus on the pituitary hormone TSH. Functional, intregrative, doctors are educated beyond and often better if you find a good one.
If you’re here to learn about GLP’s and are overweight you might have avoided if your thyroid, cortisol, sex hormones were balanced. Or at least had a better metabolism.
I am so thankful I started hormones before the rest of this.
roxygirl
GLP-1 Novice
Here are some books and websites.
Stop The Thyroid Madness. Free website and books for a fee. Very easy to understand. But don’t stop there.
Rethinking Hypothyroidism by Antonio Blanca ( former president of the American Thyroid Association) he even sees how Endo’s are not the answer.
If you’re taking NDT or T3 Liothyronnie/ Cytomel
The Thyroid Patient’s Manual by Paul Robinson and his blog YouTube are excellent.
The Facebook group Thyroid Patients Canada Support is ran by Tania S Smith. There’s no one more qualified or educated as Ms. Smith. Her articles are for Americans, Europeans, not just Canadians. She can teach us how the hormones function. You can find her articles online if fb isn’t an option. She’s a thyroid advocate and speaks all over the world for better treatment.
Social media IG Dr. Jamie Gilliam packs a lot of information in a one minute video for the visual learners. She’s hilarious too. You can book a very affordable consult with her via Zoom to sort out any concerns.
Stop The Thyroid Madness. Free website and books for a fee. Very easy to understand. But don’t stop there.
Rethinking Hypothyroidism by Antonio Blanca ( former president of the American Thyroid Association) he even sees how Endo’s are not the answer.
If you’re taking NDT or T3 Liothyronnie/ Cytomel
The Thyroid Patient’s Manual by Paul Robinson and his blog YouTube are excellent.
The Facebook group Thyroid Patients Canada Support is ran by Tania S Smith. There’s no one more qualified or educated as Ms. Smith. Her articles are for Americans, Europeans, not just Canadians. She can teach us how the hormones function. You can find her articles online if fb isn’t an option. She’s a thyroid advocate and speaks all over the world for better treatment.
Social media IG Dr. Jamie Gilliam packs a lot of information in a one minute video for the visual learners. She’s hilarious too. You can book a very affordable consult with her via Zoom to sort out any concerns.
GeforceRTX
Recently Joined
My TSH came at 8 :0 but FT3 and FT4 are in normal ranges, I cant afford a doc yet until Jan.
Can this be related to Tirze? This is my first blood work post Tirz, before that it was in normal range.
My Enzymes and Cholesterol are higher too 🙁 despite weight loss.
Can this be related to Tirze? This is my first blood work post Tirz, before that it was in normal range.
My Enzymes and Cholesterol are higher too 🙁 despite weight loss.
Thank you for the resources! I waited eight months to get in with an endocrinologist only for him to tell me to lose weight and that thyroid issues don't cause hair loss.
DM me if you want help finding a menopause specialist. 💖
It wasn't a joke. I was being serious.... I know a lot of women who went to endos that did not help them.
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I appreciate that, but I'm not quite there yet! I don't think they'd take me.
keangkong
A Not So Fat Dude
That generally means your T3 and T4 are below what they ought to be. Although the numbers for your T3 and T4 are normal for many people, your elevated TSH means that your body is saying that it needs more. Doctors rely primarily on TSH in determining whether your body is making enough T3 and T4. I'm speaking generally. Go to an endocrinologist, who will be more knowledgeable than I am on the subject. Taking too much thyroid medicine can quickly cause severe health problems. Don't try adjusting your thyroid levels yourself; doing so is more dangerous than taking grey market tirzepatide if you're too heavy. Wait until January and visit an endocrinologist.
Okay.. I get now why you laughed.
roxygirl
GLP-1 Novice
This gaslighting is inexcusable. I tell everyone who’ll listen to advocate for yourself by learning all that you can, find a good alternative doctor and stay away from end-holes.
It wasn't an insult! It would just be a bit early for me!
The problem is the waits to get in! I waited eight months for the last one. I went in armed with plenty of research! But I always appreciate new rabbit holes to dive into. 😊
roxygirl
GLP-1 Novice
Finding a well educated integrative, functional or a naturopathic ND ( that can prescribe) because Endo’s that depend on the TSH to determine what a “ Free” T4 or “ Free” T3 not a total or an uptake is negligence out of pure ignorance.
In thyroid disease and therapy, even when TSH is normalized, we can still be genuinely hypothyroid if we do not have enough “Free” T3 getting into our thyroid hormone receptors in cells throughout the body.
Most people know there’s two ways we get T3 into our cells’ nuclei:
- From circulating Free T3, and
- From circulating Free T4 hormone that is converted into T3 at a variable rate.
The body’s dependence on a baseline of healthy circulating T3 is a principle that Antonio Bianco has emphasized in numerous publications.
“ T3 and T4 cannot enter cells by passive diffusion. As they enter the cell, they must be carried on transmembrane thyroid hormone transporters, some of which have a relatively higher preference for T3 and others which have a relatively higher preference for T4.
- T3 hormone does not need to be converted. It is already in the active form, ready to bind with receptors. If it is not inactivated to T2 by D3 enzyme expressed in the cell (D3 is not shown in this diagram), a large percentage of T3 entering on transporters can be ushered directly into the nucleus.
- Circulating Free T3 fills the bottom layer of the gray sphere of nuclear receptors. Of course, nuclei don’t have “layers,” but TRs are distributed throughout the nucleus. This visually depicts the fact that each tissue depends on a baseline amount of circulating T3.
- Deiodinase type 2 (D2) enzyme activity (and in other cells, D1, not shown) converts T4 hormone locally into T3, but at a highly “variable rate” because D2 will be progressively deactivated as T4 rises within reference range. (Analogy: You can imagine that the D2 enzyme is like an office worker who gets overworked and discouraged when too much T4 paperwork gets put on his desk that requires processing.)
- T3 converted locally from T4 tops up T3 levels within the nuclear compartment. T4 is the second priority source for T3, a source that enables customization of T3 availability from tissue to tissue, as long as D2 and D1 enzymes can convert T4 locally at a healthy rate. The cell is simply not equipped to make extra T3 supply locally from converted T4 if or when FT3 supply falls short. T4 is nature’s version of “sustained-release T3” except that T3 production is highly variable.
- In this particular tissue, a certain percentage of “unoccupied receptors” is necessary for euthyroid status. If too many of the unoccupied receptors are occupied, it will create localized thyrotoxicosis. If too few are occupied, the tissue will be hypothyroid. Therefore, T3 hormone sufficiency is the ultimate determiner of euthyroid status throughout the body.
- The T3 bound to receptors will enable genomic signalling. In this cell located in the pituitary thyrotrophs, T3 will perform genetic transcription of TSH mRNA (see the arrow under the gray sphere), which, together with TRH hormone from the hypothalamus, co-regulates the level of TSH hormone secretion. In a different tissue or organ, T3 binding will signal to different genes that enable other essential biological processes to occur.
- After binding with TRs for 30 minutes to several hours, each T3 molecule exits the nucleus and returns to the cell’s cytosol (the green area in the model).
- The ratio of T3 and T4 hormones floating in the cytosol is then transported out of the cellby the same thyroid hormone transporters that brought T4 and T3 into the cell (exit transport is not shown in the diagram). The rate of hormone influx matches the rate of hormone efflux, much like breathing in and breathing out.
- This means that intracellular T3 and T4 ratios and levels directly affect FT3 and FT4 concentrations in blood. There is no “secret compartment” for T3 in the body, no “black hole” that sucks up T3 and never lets it go back into blood. There are only different rates at which each tissue exchanges hormones with blood, and one “global” rate of exchange that is comprised of the net rate of all tissues. The body converts and recycles T3 and T4 hormone among many cells and tissues until they are converted to other thyroid metabolites and/or excreted from the body”
That part is true. It is SO hard to get in with new doctors. It took me 6 months to get my husband into my GP. When I had to switch my son over from his pediatrician it didn't take as long because I asked the doctor directly at my annual.. it was only 3 months 🙄
I need to find a family dermatologist. I am dreading it....
Decreasing estrogen levels with menopause also can impair the conversion of T4 to T3 which also contributes to fatigue mood swings weight gain even though the numbers look normal. You need a good endocrinologist and one that will listen to your symptoms
